2022-03-102022-03-1020171663-2818https://hdl.handle.net/11424/221966enginfo:eu-repo/semantics/closedAccess17OH-PREGNENOLONE SEEMS A MAJOR DRIVE OF ANDROGEN EXCESS IN PATIENTS WITH 11 beta-HYDROXYLASE DEFICIENCYconferenceObjectWOS:000412595401100doiWOS:0004125954011001663-2826