Publication:
Nutrients in schizophrenia: A focus on the pathophysiological pathway

dc.contributor.authorGÜNEŞ, FATMA ESRA
dc.contributor.authorsAkduman G., Kurtbeyoglu E., Gunes F.E.
dc.date.accessioned2022-03-15T02:17:05Z
dc.date.available2022-03-15T02:17:05Z
dc.date.issued2021
dc.description.abstractObjectives The aim of this article is to explain the nutrients that play an active role in the pathophysiology of schizophrenia. Methods This paper is a narrative literature review of relevant articles and prior works that have been central to the topic including the active nutrients in the pathophysiology of schizophrenia. Results The findings are compiled under six headings. The changes in the antioxidant defense system, dopamine pathway, serotonin pathway, gamma-aminobutyric acid (GABA) pathway, glutamate pathway, the endocannabinoid system, and metabolomic profile were investigated in relation to nutrients. Conclusions This review provides an update of scientific knowledge on the growing role of nutrition in schizophrenia. Nutrient deficiencies that occur frequently in these patients should be followed and eliminated to ensure the correct functioning of the pathophysiological pathways of the disease. © 2021 Pacini Editore S.p.A.. All rights reserved.
dc.identifier.doi10.36148/2284-0249-424
dc.identifier.issn22840249
dc.identifier.urihttps://hdl.handle.net/11424/248279
dc.language.isoeng
dc.publisherPacini Editore S.p.A./AU-CNS
dc.relation.ispartofJournal of Psychopathology
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectEndocannabinoids
dc.subjectNeurotransmitter agents
dc.subjectNutrients
dc.subjectSchizophrenia
dc.titleNutrients in schizophrenia: A focus on the pathophysiological pathway
dc.typereview
dspace.entity.typePublication
local.avesis.idadc24735-da34-4c81-b1dd-d4d34f69ee61
local.import.packageSS21
local.import.sourceScopus
local.indexed.atSCOPUS
oaire.citation.endPage186
oaire.citation.issue3
oaire.citation.startPage181
oaire.citation.titleJournal of Psychopathology
oaire.citation.volume27
relation.isAuthorOfPublicationfdd3ef33-8958-4d6f-9295-d2a9647cd236
relation.isAuthorOfPublication.latestForDiscoveryfdd3ef33-8958-4d6f-9295-d2a9647cd236

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