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Sepsis-Induced Myocardial Depression and Takotsubo Syndrome

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dc.contributor.authorCİNEL, İSMAİL HAKKI
dc.contributor.authorsArslantas, Mustafa Kemal; Altun, Gulbin Tore; Cinel, Ismail
dc.date.accessioned2022-03-10T11:39:34Z
dc.date.available2022-03-10T11:39:34Z
dc.date.issued2015-08-05
dc.description.abstractSepsis induced temporary myocardial dysfunction characterized as impairment of myocardial contraction is an important cause of mortality and morbidity in intensive care units. Takotsubo syndrome (TS) is temporary ballooning and dysfunction of the apical part of left ventricle without significant stenosis of coronary arteries. Recently, it was suggested that impairment in regional catecholamine distribution caused by stress factors and excessive cardiac sympathetic activity mechanism play role in sepsis such as other causes of TS. Additionally, vasopressor agents (as noradrenaline) which are widely used in sepsis treatment may be triggering factor. Serial case reports of sepsis associated TS are reported, however pathophysiology, diagnosis and treatment strategies of these two different syndromes is not obvious.
dc.identifier.doi10.4274/tybdd.85547
dc.identifier.eissn2147-267X
dc.identifier.issn2146-6416
dc.identifier.urihttps://hdl.handle.net/11424/219883
dc.identifier.wosWOS:000422270200001
dc.language.isotur
dc.publisherGALENOS YAYINCILIK
dc.relation.ispartofJOURNAL OF THE TURKISH SOCIETY OF INTENSIVE CARE-TURK YOGUN BAKM DERNEGI DERGISI
dc.rightsinfo:eu-repo/semantics/openAccess
dc.subjectSepsis
dc.subjectTakotsubo syndrome
dc.subjectmyocardial dysfunction
dc.titleSepsis-Induced Myocardial Depression and Takotsubo Syndrome
dc.typereview
dspace.entity.typePublication
local.avesis.id83623229-36f4-41c0-ba48-ab3368eddf71
local.import.packageSS4
local.indexed.atWOS
local.indexed.atTRDIZIN
local.journal.numberofpages8
oaire.citation.endPage56
oaire.citation.issue2
oaire.citation.startPage49
oaire.citation.titleJOURNAL OF THE TURKISH SOCIETY OF INTENSIVE CARE-TURK YOGUN BAKM DERNEGI DERGISI
oaire.citation.volume13
relation.isAuthorOfPublication202e7185-bae9-46db-9cdd-f2f0881870aa
relation.isAuthorOfPublication.latestForDiscovery202e7185-bae9-46db-9cdd-f2f0881870aa

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