Publication:
Proteasome and Neurodegenerative Diseases

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dc.contributor.authorsCatalgol, Betul; Grune, Tilman
dc.contributor.editorGrune, T
dc.date.accessioned2022-03-11T21:33:42Z
dc.date.accessioned2026-01-10T19:14:24Z
dc.date.available2022-03-11T21:33:42Z
dc.date.issued2012
dc.description.abstractProteasomal degradation of damaged proteins is involved in the pathogenesis of many neurodegenerative diseases including Alzheimer's, Parkinson's, Huntington's, stroke, and amyotrophic lateral sclerosis. A malfunction of the proteasomal activity may be the result or the consequence of protein aggregation, which is a key process for most neurodegenerative diseases. Because of the widespread aspects of the proteasomal involvement in the progression of these diseases, many studies are focused on this research.
dc.identifier.doi10.1016/B978-0-12-397863-9.00011-0
dc.identifier.isbn978-0-12-397863-9
dc.identifier.issn1877-1173
dc.identifier.pubmed22727428
dc.identifier.urihttps://hdl.handle.net/11424/222771
dc.identifier.wosWOS:000306933300012
dc.language.isoeng
dc.publisherELSEVIER ACADEMIC PRESS INC
dc.relation.ispartofPROTEASOMAL SYSTEM IN AGING AND DISEASE
dc.relation.ispartofseriesProgress in Molecular Biology and Translational Science
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectAMYOTROPHIC-LATERAL-SCLEROSIS
dc.subjectOXIDATIVELY MODIFIED PROTEINS
dc.subjectNEURONAL INTRANUCLEAR INCLUSIONS
dc.subjectSUPEROXIDE-DISMUTASE PROTEINS
dc.subjectALPHA-SYNUCLEIN AGGREGATION
dc.subjectG93A-SOD1 TRANSGENIC MICE
dc.subjectAMYLOID-BETA-PROTEIN
dc.subjectALZHEIMERS-DISEASE
dc.subjectPARKINSONS-DISEASE
dc.subjectLIPID-PEROXIDATION
dc.titleProteasome and Neurodegenerative Diseases
dc.typebookPart
dspace.entity.typePublication
oaire.citation.endPage414
oaire.citation.startPage397
oaire.citation.titlePROTEASOMAL SYSTEM IN AGING AND DISEASE
oaire.citation.volume109

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