Publication:
Disease Mechanisms

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dc.contributor.authorsDireskeneli, Haner; Saruhan-Direskeneli, Guher
dc.contributor.editorYazici, Y
dc.contributor.editorHatemi, G
dc.contributor.editorSeyahi, E
dc.contributor.editorYazici, H
dc.date.accessioned2022-03-11T21:34:04Z
dc.date.accessioned2026-01-11T06:16:37Z
dc.date.available2022-03-11T21:34:04Z
dc.date.issued2020
dc.description.abstractBehcet syndrome (BS) is a systemic inflammatory disorder with a diverse spectrum of clinical manifestations including mucocutaneous, ocular, vascular, gastrointestinal, musculoskeletal, and central nervous system involvement [1]. A complex genetic background leading to a pro-inflammatory, innate immune system-derived activation perpetuated by adaptive immune responses against environmental antigens or/and autoantigens is accepted as the main pathogenic mechanism in BS [2, 3].
dc.identifier.bookdoi10.1007/978-3-030-24131-5
dc.identifier.doi10.1007/978-3-030-24131-5_15
dc.identifier.isbn978-3-030-24131-5; 978-3-030-24130-8
dc.identifier.urihttps://hdl.handle.net/11424/222904
dc.identifier.wosWOS:000577078500015
dc.language.isoeng
dc.publisherSPRINGER INTERNATIONAL PUBLISHING AG
dc.relation.ispartofBEHCET SYNDROME, 2ND EDITION
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectREGULATORY T-CELLS
dc.subjectNATURAL-KILLER-CELLS
dc.subjectDAGGER-ETS DISEASE
dc.subjectFAMILIAL MEDITERRANEAN FEVER
dc.subjectMONOSODIUM URATE CRYSTALS
dc.subjectBLOOD MONONUCLEAR-CELLS
dc.subjectOCULAR BEHCETS-DISEASE
dc.subjectHEAT-SHOCK PROTEINS
dc.subjectPERIPHERAL-BLOOD
dc.subjectIMMUNE-RESPONSE
dc.titleDisease Mechanisms
dc.typebookPart
dspace.entity.typePublication
oaire.citation.endPage222
oaire.citation.startPage209
oaire.citation.titleBEHCET SYNDROME, 2ND EDITION

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